cell injuryの例文

• Schnellmann, R . G . : Pathophysiology of Nephrotoxic Cell Injury.
• Research is organised around major research projects within the overall theme of Liver Cell Injury and Repair.
• Roche say it is likely most cases of liver cell injury can be prevented by attention to patient symptoms.
• These inflammatory responses cause intracranial pressure, pressure which leads to cell injury and in some situations cell death.
• Cummings, B . S . and Schnellmann, R . G . : Pathophysiology of Nephrotoxic Cell Injury.
• Cummings, B . S . and Schnellman, R . G . : Pathophysiology of Nephrotoxic Cell Injury.
• Glycyrrhizin inhibits liver cell injury and is given intravenously for the treatment of chronic viral hepatitis and cirrhosis in Japan.
• These inflammatory responses cause increased intracranial pressure, which leads to cell injury and in some situations, cell death.
• Atherosclerosis has been conceptualized as a chronic inflammatory response to endothelial cell injury and dysfunction possibly arising from chronic dental infection.
• These include drugs that prevent blood coagulation, as well as new therapies to prevent nerve cell injury like calcium channel blockers and neurotransmitter inhibitors.
• Any poisoning of the cell from any source increase the likelihood of a cancer to erupt as any cell injury can stimulate its uncontrolled division.
• It is, therefore, this lower-energy form of singlet oxygen ( 1?g ) that is implicated in cell injury and cell death.
• Current areas of research include viral hepatitis, liver cancer, liver inflammation and metabolism, liver cell injury and metabonomic and proteomic studies ."
• His research has centered on the cell biology of acute kidney injury with an emphasis on proximal tubule cell injury secondary to ischemia and / or nephrotoxins.
• Structural signs that indicate irreversible cell injury and the progression of necrosis include dense clumping and progressive disruption of genetic material, and disruption to membranes of cells and organelles.
• He contributed to histochemistry, the study of the chemical components of cells and tissues, and carried out research in the areas of cell injury, inflammation and wound healing.
• First noted is infiltration by lymphocytes, followed by epithelial cell injury, then inflammatory lesions and recruitment of fibroblasts and myofibroblasts, which proliferate and secrete proteins forming scar tissue.
• The function of inflammation is to eliminate the initial cause of cell injury, clear out necrotic cells and tissues damaged from the original insult and the inflammatory process, and to initiate tissue repair.
• When host cells die, either by programmed cell death ( also called apoptosis ) or by cell injury due to a bacterial or viral infection, phagocytic cells are responsible for their removal from the affected site.
• He and his colleague, Dr . Virginia M .-Y . Lee, favor the second hypothesis, that cell injury and death occurs from the tangles that build up inside cells when a crucial internal protein goes awry.